Abstract
Gonadotropin-releasing hormone is implicated in cognitive functions, and its loss is a factor in pathological brain ageing. There are similarities between these processes and the neurological and cognitive deficits observed in patients with long COVID. Here, we explore the hypothesis that neuroanatomical and transcriptomic alterations associated with long COVID could stem from this neuroendocrine perturbation.
| Original language | English |
|---|---|
| Journal | Nature Reviews Endocrinology |
| Volume | 20 |
| Issue number | 4 |
| Pages (from-to) | 189-190 |
| Number of pages | 2 |
| ISSN | 1759-5029 |
| DOIs | |
| Publication status | Published - 04.2024 |
Funding
| Funders | Funder number |
|---|---|
| European Research Council ERC-Synergy-Grant-2019-WATCH | 810331 |
| Agence Nationale de la Recherche en Santé et la Fondation pour la Recherche Médicale | ECTZ200878 |
| Horizon 2020 | 847941 miniNO |
UN SDGs
This output contributes to the following UN Sustainable Development Goals (SDGs)
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SDG 3 Good Health and Well-being
Research Areas and Centers
- Academic Focus: Center for Brain, Behavior and Metabolism (CBBM)
DFG Research Classification Scheme
- 2.22-09 Pharmacology
- 2.22-17 Endocrinology, Diabetology, Metabolism
- 2.23-08 Human Cognitive and Systems Neuroscience
Coronavirus related work
- Research on SARS-CoV-2 / COVID-19
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