Pontine lesions may cause selective deficits of "slow" vergence eye movements

H. Rambold; G. Neumann; T. Sander; C. Helmchen

doi:10.1196/annals.1325.069

Pontine lesions may cause selective deficits of "slow" vergence eye movements

H. Rambold^*, G. Neumann, T. Sander, C. Helmchen

^*Corresponding author for this work

10 Citations (Scopus)

Abstract

To address the role of pontine nuclei in vergence control, eye movements to ramp ('slow vergence') and step targets ('fast vergence') were recorded in two patients with unilateral mediolateral pontine infarctions and in ten healthy controls. 'Slow' vergence and conjugate smooth pursuit eye movements were impaired while 'fast' vergence was not. We conclude that like smooth pursuit signals, vergence signals are distributed in the pontine nuclei.

Original language	English
Journal	Annals of the New York Academy of Sciences
Volume	1039
Pages (from-to)	567-570
Number of pages	4
ISSN	0077-8923
DOIs	https://doi.org/10.1196/annals.1325.069
Publication status	Published - 01.01.2005

Research Areas and Centers

Academic Focus: Center for Brain, Behavior and Metabolism (CBBM)

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This output contributes to the following UN Sustainable Development Goals (SDGs)

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10.1196/annals.1325.069

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AB - To address the role of pontine nuclei in vergence control, eye movements to ramp ('slow vergence') and step targets ('fast vergence') were recorded in two patients with unilateral mediolateral pontine infarctions and in ten healthy controls. 'Slow' vergence and conjugate smooth pursuit eye movements were impaired while 'fast' vergence was not. We conclude that like smooth pursuit signals, vergence signals are distributed in the pontine nuclei.

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Pontine lesions may cause selective deficits of "slow" vergence eye movements

Abstract

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