Non-compaction-kardiomyopathie

Heinrich Wieneke; Till Neumann; Frank Breuckmann; Peter Hunold; Jochen W.U. Fries; Olaf Dirsch; Raimund Erbel

doi:10.1007/s00059-005-2734-1

Non-compaction-kardiomyopathie

Translated title of the contribution: Isolated left ventricular non-compaction

Heinrich Wieneke^*, Till Neumann, Frank Breuckmann, Peter Hunold, Jochen W.U. Fries, Olaf Dirsch, Raimund Erbel

^*Corresponding author for this work

Clinic of Radiology and Nuclear Medicine

5 Citations (Scopus)

Abstract

Isolated non-compaction of the ventricular myocardium (INVM), also known as left ventricular hypertrabeculation or spongy myocardium, belongs to the "unclassified" cardiomyopathies according to the World Health Organization. The main characteristic of this entity is a prominent trabeculation of the left ventricle with deep intertrabecular recesses communicating with the ventricular cavity. The pathomechanism of INVM is thought to be an arrest in cardiac myogenesis with persistence of embryonic myocardial morphology. The most frequent clinical manifestations include congestive heart failure, ventricular arrhythmias and systemic thromboembolic events. The therapy of INVM comprises standard medical therapy for heart failure.

Translated title of the contribution	Isolated left ventricular non-compaction
Original language	German
Journal	Herz
Volume	30
Issue number	6
Pages (from-to)	571-574
Number of pages	4
ISSN	0340-9937
DOIs	https://doi.org/10.1007/s00059-005-2734-1
Publication status	Published - 01.09.2005

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10.1007/s00059-005-2734-1

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title = "Non-compaction-kardiomyopathie",

abstract = "Isolated non-compaction of the ventricular myocardium (INVM), also known as left ventricular hypertrabeculation or spongy myocardium, belongs to the {"}unclassified{"} cardiomyopathies according to the World Health Organization. The main characteristic of this entity is a prominent trabeculation of the left ventricle with deep intertrabecular recesses communicating with the ventricular cavity. The pathomechanism of INVM is thought to be an arrest in cardiac myogenesis with persistence of embryonic myocardial morphology. The most frequent clinical manifestations include congestive heart failure, ventricular arrhythmias and systemic thromboembolic events. The therapy of INVM comprises standard medical therapy for heart failure.",

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T1 - Non-compaction-kardiomyopathie

AU - Wieneke, Heinrich

AU - Neumann, Till

AU - Breuckmann, Frank

AU - Hunold, Peter

AU - Fries, Jochen W.U.

AU - Dirsch, Olaf

AU - Erbel, Raimund

PY - 2005/9/1

Y1 - 2005/9/1

N2 - Isolated non-compaction of the ventricular myocardium (INVM), also known as left ventricular hypertrabeculation or spongy myocardium, belongs to the "unclassified" cardiomyopathies according to the World Health Organization. The main characteristic of this entity is a prominent trabeculation of the left ventricle with deep intertrabecular recesses communicating with the ventricular cavity. The pathomechanism of INVM is thought to be an arrest in cardiac myogenesis with persistence of embryonic myocardial morphology. The most frequent clinical manifestations include congestive heart failure, ventricular arrhythmias and systemic thromboembolic events. The therapy of INVM comprises standard medical therapy for heart failure.

AB - Isolated non-compaction of the ventricular myocardium (INVM), also known as left ventricular hypertrabeculation or spongy myocardium, belongs to the "unclassified" cardiomyopathies according to the World Health Organization. The main characteristic of this entity is a prominent trabeculation of the left ventricle with deep intertrabecular recesses communicating with the ventricular cavity. The pathomechanism of INVM is thought to be an arrest in cardiac myogenesis with persistence of embryonic myocardial morphology. The most frequent clinical manifestations include congestive heart failure, ventricular arrhythmias and systemic thromboembolic events. The therapy of INVM comprises standard medical therapy for heart failure.

UR - https://www.scopus.com/pages/publications/25444444720

U2 - 10.1007/s00059-005-2734-1

DO - 10.1007/s00059-005-2734-1

M3 - Zeitschriftenaufsätze

C2 - 16170691

AN - SCOPUS:25444444720

SN - 0340-9937

VL - 30

SP - 571

EP - 574

JO - Herz

JF - Herz

IS - 6

ER -

Non-compaction-kardiomyopathie

Abstract

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