Increased co-expression of the natural killer cell receptor NKG2D and further natural killer cell receptors on CD4⁺ T cells in granulomatosis with polyangiitis

S Schüler; S Wolters; S Pitann; D Kabelitz; P Lamprecht

Increased co-expression of the natural killer cell receptor NKG2D and further natural killer cell receptors on CD4⁺ T cells in granulomatosis with polyangiitis

S Schüler, S Wolters, S Pitann, D Kabelitz, P Lamprecht

Clinic of Rheumatology

University of Kiel

Original language	English
Journal	Clinical and Experimental Rheumatology
Volume	33
Issue number	2 Suppl 89
Pages (from-to)	S-183-4
ISSN	0392-856X
Publication status	Published - 29.05.2015

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This output contributes to the following UN Sustainable Development Goals (SDGs)

Cite this

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title = "Increased co-expression of the natural killer cell receptor NKG2D and further natural killer cell receptors on CD4⁺ T cells in granulomatosis with polyangiitis",

author = "S Sch{\"u}ler and S Wolters and S Pitann and D Kabelitz and P Lamprecht",

year = "2015",

month = may,

day = "29",

language = "English",

volume = "33",

pages = "S--183--4",

journal = "Clinical and Experimental Rheumatology",

issn = "0392-856X",

number = "2 Suppl 89",

}

Increased co-expression of the natural killer cell receptor NKG2D and further natural killer cell receptors on CD4⁺ T cells in granulomatosis with polyangiitis. / Schüler, S; Wolters, S; Pitann, S et al.
In: Clinical and Experimental Rheumatology, Vol. 33, No. 2 Suppl 89, 29.05.2015, p. S-183-4.

Research output: Journal Articles › Journal articles › Research › peer-review

TY - JOUR

T1 - Increased co-expression of the natural killer cell receptor NKG2D and further natural killer cell receptors on CD4⁺ T cells in granulomatosis with polyangiitis

AU - Schüler, S

AU - Wolters, S

AU - Pitann, S

AU - Kabelitz, D

AU - Lamprecht, P

PY - 2015/5/29

Y1 - 2015/5/29

M3 - Journal articles

C2 - 26016770

SN - 0392-856X

VL - 33

SP - S-183-4

JO - Clinical and Experimental Rheumatology

JF - Clinical and Experimental Rheumatology

IS - 2 Suppl 89

ER -