Heart failure and cardiac involvement as isolated manifestation of familial form of transthyretin amyloidosis resulting from val30met mutation with no clinical signs of polyneuropathy

Daniel C. Christoph; Dirk Boese; Kristian T.M. Johnson; Thomas W. Schlosser; Peter Hunold; Hideo A. Baba; Raimund Erbel; Sebastian Philipp

doi:10.1161/CIRCHEARTFAILURE.109.853697

Heart failure and cardiac involvement as isolated manifestation of familial form of transthyretin amyloidosis resulting from val30met mutation with no clinical signs of polyneuropathy

Daniel C. Christoph, Dirk Boese, Kristian T.M. Johnson, Thomas W. Schlosser, Peter Hunold, Hideo A. Baba, Raimund Erbel, Sebastian Philipp^*

^*Corresponding author for this work

Clinic of Radiology and Nuclear Medicine

2 Citations (Scopus)

Original language	English
Journal	Circulation: Heart Failure
Volume	2
Issue number	5
Pages (from-to)	512-515
Number of pages	4
ISSN	1941-3289
DOIs	https://doi.org/10.1161/CIRCHEARTFAILURE.109.853697
Publication status	Published - 01.09.2009

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10.1161/CIRCHEARTFAILURE.109.853697

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Christoph, D. C., Boese, D., Johnson, K. T. M., Schlosser, T. W., Hunold, P., Baba, H. A., Erbel, R., & Philipp, S. (2009). Heart failure and cardiac involvement as isolated manifestation of familial form of transthyretin amyloidosis resulting from val30met mutation with no clinical signs of polyneuropathy. Circulation: Heart Failure, 2(5), 512-515. https://doi.org/10.1161/CIRCHEARTFAILURE.109.853697

@article{092bcd3a79584bf2bc15af49278dd715,

title = "Heart failure and cardiac involvement as isolated manifestation of familial form of transthyretin amyloidosis resulting from val30met mutation with no clinical signs of polyneuropathy",

author = "Christoph, {Daniel C.} and Dirk Boese and Johnson, {Kristian T.M.} and Schlosser, {Thomas W.} and Peter Hunold and Baba, {Hideo A.} and Raimund Erbel and Sebastian Philipp",

year = "2009",

month = sep,

day = "1",

doi = "10.1161/CIRCHEARTFAILURE.109.853697",

language = "English",

volume = "2",

pages = "512--515",

journal = "Circulation: Heart Failure",

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Christoph, DC, Boese, D, Johnson, KTM, Schlosser, TW, Hunold, P, Baba, HA, Erbel, R & Philipp, S 2009, 'Heart failure and cardiac involvement as isolated manifestation of familial form of transthyretin amyloidosis resulting from val30met mutation with no clinical signs of polyneuropathy', Circulation: Heart Failure, vol. 2, no. 5, pp. 512-515. https://doi.org/10.1161/CIRCHEARTFAILURE.109.853697

Heart failure and cardiac involvement as isolated manifestation of familial form of transthyretin amyloidosis resulting from val30met mutation with no clinical signs of polyneuropathy. / Christoph, Daniel C.; Boese, Dirk; Johnson, Kristian T.M. et al.
In: Circulation: Heart Failure, Vol. 2, No. 5, 01.09.2009, p. 512-515.

Research output: Journal Articles › Journal articles › Research › peer-review

TY - JOUR

T1 - Heart failure and cardiac involvement as isolated manifestation of familial form of transthyretin amyloidosis resulting from val30met mutation with no clinical signs of polyneuropathy

AU - Christoph, Daniel C.

AU - Boese, Dirk

AU - Johnson, Kristian T.M.

AU - Schlosser, Thomas W.

AU - Hunold, Peter

AU - Baba, Hideo A.

AU - Erbel, Raimund

AU - Philipp, Sebastian

PY - 2009/9/1

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DO - 10.1161/CIRCHEARTFAILURE.109.853697

M3 - Journal articles

C2 - 19808383

AN - SCOPUS:70350457662

SN - 1941-3289

VL - 2

SP - 512

EP - 515

JO - Circulation: Heart Failure

JF - Circulation: Heart Failure

IS - 5

ER -

Heart failure and cardiac involvement as isolated manifestation of familial form of transthyretin amyloidosis resulting from val30met mutation with no clinical signs of polyneuropathy

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