Dysregulated complement activation during acute myocardial infarction leads to endothelial glycocalyx degradation and endothelial dysfunction via the C5a:C5a-Receptor1 axis

Carl Vahldieck, Samuel Löning, Constantin Hamacher, Benedikt Fels, Bettina Rudzewski, Laura Nickel, Joachim Weil, Henry Nording, Lasse Baron, Marie Kleingarn, Christian Marcel Karsten, Kristina Kusche-Vihrog

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Biochemistry, Genetics and Molecular Biology