Organisation profile
About us
The Institute of Neurogenetics focuses its clinical and research activities on hereditary forms of movement disorders, mainly Parkinson disease and dystonia. Discovering the genetic factors causing or contributing to movement disorders is the Institute’s main aim of research.
With an interdisciplinary team of movement disorder specialists, clinician and basic scientists, study nurses and research technologists, PhD and medical students closely collaborating, the research is translational and interdisciplinary in nature and directed from the patient to the bench and back.
Career development and teaching are an important and integral part of the Institute’s work. Neurology residents participate in structured movement disorders and neurogenetics fellowships; Bachelor, Master, and PhD students in the field of Molecular Life Sciences are trained at the Institute.
Research Areas and Centers
- Research Area: Medical Genetics
- Centers: Center for Rare Diseases (ZSE)
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Collaborations and top research areas from the last five years
Profiles
Research output
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Convergence Deficits in Myoclonus-Dystonia Point to Cerebellar Impairment
Gerkensmeier, S., Bolte, C., Radecke, J. O., Hamami, F., Sprenger, A., Helmchen, C., Chen, R., Callister, M., Cortez Grippe, T., Klein, C., Brüggemann, N., Bäumer, T., Münchau, A. & Weissbach, A., 2026, In: Movement Disorders Clinical Practice.Research output: Journal Articles › Journal articles › Research › peer-review
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Somatic genomic and environmental modifiers in Parkinson’s disease and related disorders
Trinh, J. (Principal Investigator (PI))
01.01.25 → 31.12.29
Project: DFG Individual Projects › DFG Fellowships: Heisenberg Programme
Prizes
Datasets
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Multivariate GWAS of Alzheimer’s disease CSF biomarker profiles implies GRIN2D in synaptic functioning
Neumann, A. (Scientific Creator), Ohlei, O. (Scientific Creator), Küçükali, F. (Scientific Creator), Bos, I. J. (Scientific Creator), Timsina, J. (Scientific Creator), Vos, S. (Scientific Creator), Prokopenko, D. (Scientific Creator), Tijms, B. M. (Scientific Creator), Andreasson, U. (Scientific Creator), Blennow, K. (Scientific Creator), Vandenberghe, R. (Scientific Creator), Scheltens, P. (Scientific Creator), Teunissen, C. E. (Scientific Creator), Engelborghs, S. (Scientific Creator), Frisoni, G. B. (Scientific Creator), Blin, O. (Scientific Creator), Richardson, J. C. (Scientific Creator), Bordet, R. (Scientific Creator), Lleó, A. (Scientific Creator), Alcolea, D. (Scientific Creator), Popp, J. (Scientific Creator), Marsh, T. W. (Scientific Creator), Gorijala, P. (Scientific Creator), Clark, C. (Scientific Creator), Peyratout, G. (Scientific Creator), Martinez-Lage, P. (Scientific Creator), Tainta, M. (Scientific Creator), Dobson, R. J. B. (Scientific Creator), Legido-Quigley, C. (Scientific Creator), Van Broeckhoven, C. (Scientific Creator), Tanzi, R. E. (Scientific Creator), ten Kate, M. (Scientific Creator), Lill, C. M. (Scientific Creator), Barkhof, F. (Scientific Creator), Cruchaga, C. (Scientific Creator), Lovestone, S. (Scientific Creator), Streffer, J. (Scientific Creator), Zetterberg, H. (Scientific Creator), Visser, P. J. (Scientific Creator), Sleegers, K. (Scientific Creator) & Bertram, L. (Scientific Creator), figshare, 2024
DOI: 10.6084/m9.figshare.c.6866395, https://springernature.figshare.com/collections/Multivariate_GWAS_of_Alzheimer_s_disease_CSF_biomarker_profiles_implies_GRIN2D_in_synaptic_functioning/6866395
Dataset