Single Heterozygous ATP13A2 Mutations Cause Cellular Dysfunction Associated with Parkinson's Disease

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*Corresponding authors: Dr. Carolyn M. Sue, Department of Neurogenetics, Kolling Institute, Royal North Shore Hospital and University of Sydney, St Leonard’s, NSW 2065, Australia; [email protected] and Dr. Jin-Sung Park, Department of Neurogenetics, Kolling Institute, Royal North Shore Hospital and University of Sydney, St Leonard’s, NSW 2065, Australia; [email protected] Funding agencies: This study was funded by a Parkinson’s NSW seed grant, grants from the National Health and Medical Research Council (APP1008433 and 1010839), the German Research Foundation (FOR2488), and the Hermann and Lilly Schilling Foundation. Acknowledgments: J.-S.P. is a recipient of a Parkinson’s NSW seed grant. C.M.S was awarded funding from the National Health and Medical Research Council (APP1008433 and 1010839), Parkinson’s NSW, Ramsay Research and Teaching fund and the Australian Mitochondrial Disease Foundation. CK receives funding from the DFG (FOR2488, P2) and the Hermann and Lilly Schilling Foundation.