Mitochondrial C5aR1 activity in macrophages controls IL-1β production underlying sterile inflammation

Nathalie Niyonzima, Jubayer Rahman, Natalia Kunz, Erin E. West, Tilo Freiwald, Jigar V. Desai, Nicolas S. Merle, Alexandre Gidon, Bjørnar Sporsheim, Michail S. Lionakis, Kristin Evensen, Beate Lindberg, Karolina Skagen, Mona Skjelland, Parul Singh, Markus Haug, Marieta M. Ruseva, Martin Kolev, Jack Bibby, Olivia MarshallBrett O’Brien, Nigel Deeks, Behdad Afzali, Richard J. Clark, Trent M. Woodruff, Milton Pryor, Zhi Hong Yang, Alan T. Remaley, Tom E. Mollnes, Stephen M. Hewitt, Bingyu Yan, Majid Kazemian, Máté G. Kiss, Christoph J. Binder, Bente Halvorsen, Terje Espevik^*, Claudia Kemper^*

^*Korrespondierende/r Autor/-in für diese Arbeit

Institut für Anatomie
Institut für Systemische Entzündungsforschung

Nord-Trøndelag Health Trust
National Heart, Lung and Blood Institute
National Institute of Diabetes and Digestive and Kidney Diseases
National Institute on Aging
Trondheim University Hospital
Drammen Hospital
Rikshospitalet-Radiumhospitalet HF
University of Oslo
GlaxoSmithKline
The University of Queensland
University of Tromsø
National Cancer Institute
Purdue University
University of Vienna
CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences

Mitochondrial C5aR1 activity in macrophages controls IL-1β production underlying sterile inflammation

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Biochemistry, Genetics and Molecular Biology

Immunology and Microbiology

Medicine and Dentistry